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Does all this describe what happens to victims of MS, autism, chronic pain, and even heart disease?
We know for sure that high levels of ICs are found in patients who have rheumatoid arthritis, lupus, hepatitis, multiple sclerosis, ulcerative colitis (inflamed colon), certain cancers, and more. We also know that successful treatment of these diseases goes hand in glove with a reduction in IC levels.

DeFelice cites 14 studies that show inflammatory and autoimmune diseases improved when IC levels were reduced. (KD, page 195)

In a study of 83 “fully evaluated” MS patients, 85% got better with enzyme therapy (specifically, protein-eating enzymes, as Chapter Eleven will explain). Well over half of these 83 patients showed “substantial improvement.” (KD, page 196)

Researchers believe the KNOWN effectiveness of enzyme therapy in treating inflammation and autoimmune diseases may be due to the ability of enzymes to devour ICs and interrupt the immune system’s “death squad” — the inflammatory cascade of nine attackers.

What causes an IC overload in the first place, followed by the destructive autoimmune response? We don’t know for sure. Environmental pollution, drugs, certain infections, leaky gut syndrome and undigested food particles in the blood stream — all could play a role. Our genes may be another reason. It’s known that women are far more likely than men to suffer from chronic autoimmune diseases.

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i have had abdominal pain all day and wen to the bathroom 16 times today. i do this alot. my dr is checking me for crohns/colitis. my sister is a nurse and thinks it is only ibs. i also have hot flashes, fevers, night sweats, etc. i had an anal fissure when i was younger and my bowels didnt form untili was four. is this more than ibs.

Symptoms of IBS are abdominal pain or discomfort associated with changes in bowel habits in the absence of any structural abnormality. Colonic hypersensitivity is a sensitive but less specific sign of Irritable Bowel Syndrome. Depending on the kind of discomfort and bowel habits, IBS is also known as spastic colon, and can be subclassified into diarrhea-predominant (IBS-D), constipation-predominant (IBS-C) and Irritable Bowel Syndrome with alternating stool pattern (IBS-A). Typical is the overlap of Irritable Bowel Syndrome with chronic pelvic pain (this is probably due to misdiagnosis by the gynaecologist), fibromyalgia and mental disorder.

According to the Rome II consensus conference of the American Gastroenterological Association and international medical societies on functional bowel disorders, the diagnosis of Irritable Bowel Syndrome can be made when the following criteria are fulfilled: At least 12 weeks, which need not be consecutive, in the preceding 12 months of abdominal discomfort or pain that has 2 of 3 features:

- Relieved with defecation; and/or
- Onset associated with a change in frequency of stool; and/or
- Onset associated with a change in form (appearance) of stool. Symptoms that cumulatively support the diagnosis of IBS
- Abnormal stool frequency (for research purposes, “abnormal” may be defined as greater than 3 bowel movements per day and less than 3 bowel movements per week);
- Abnormal stool form (lumpy/hard or loose/watery stool);
- Abnormal stool passage (straining, urgency, or feeling of incomplete evacuation);
- Passage of mucus;
- Bloating or feeling of abdominal distention.

The diagnosis of a functional bowel disorder always presumes the absence of a structural or biochemical explanation for the symptoms. This has to be excluded carefully via:

- colonoscopy
- esophagogastroduodenoscopy (EGD)
- abdominal ultrasound
- blood tests: full blood count, liver enzymes, electrolytes, renal function
- stool chemistry (e.g. tests for exocrine pancreas insufficiency and other malabsorption conditions), stool microbiology, fecal fat
- H2-tests for lactose intolerance and fructose malabsorption
- deep duodenal biopsy or blood tests for celiac disease

Crohn’s disease is a chronic inflammatory disease of the digestive tract and it can involve any part of it – from the mouth to the anus. Crohn’s disease typically affects the terminal ileum as well as demarcated areas of large bowel, with other areas of the bowel being relatively unaffected. Crohn’s disease is often associated with auto-immune disorders outside the bowel, such as aphthous stomatitis and rheumatoid arthritis. Crohn’s disease should not be confused with a non-progressive and non-degenerative digestive disorder called irritable bowel syndrome. IBS is not an autoimmune disease. Ulcerative colitis is a sibling autoimmune disease to Crohn’s disease but only impacts the colon while Crohn’s disease can impact any part of the digestive tract. Furthermore, Crohn’s disease tends to impact multiple layers of the bowel lining which can lead to many additional and hard to treat complications.

Crohn’s disease patients typically suffer from chronic diarrhea and disrupted digestion, making it difficult for sufferers in the acute phase of the disease to eat and/or digest food. The inflammation of Crohn’s disease can be extremely painful and debilitating. Other common complications of Crohn’s disease include fistulas of the colon, hemorrhoids, lipid absorption problems, and anemia. Bleeding is seen in 20% cases of Crohn’s bisease, against 98% cases in ulcerative colitis.

I have this I’m told. No physical signs in colonoscopy but related mild symptoms. Blood test reveals that its "in there" Doc says. Anyone know what this blood test he is talking about is?

Unless there is some new test of which I’m unaware (doubtful)… there isn’t one specific blood test that tests for ulcerative colitis (UC). There are blood tests that show things that may be associated with UC like being anemic (low blood count that could indicate bleeding in your colon) or having a high white blood cell count or high ESR (erythrocyte sedimentation rate) that may indicate the presence of inflammation…and in UC there is lots of inflammation in your colon.

The doctor uses these results in conjunction with an internal exam of your colon (sigmoidoscopy or colonoscopy), examination of your stool for blood, and a barium enema xray to make the diagnosis.

So…the blood tests alone won’t tell if you have UC. It’s like putting all the puzzle pieces together and the picture you finally get is UC.

if so what have you found to be effective as a treatment? anyone gone into long term remission? i have a friend with the problem and were trying to get some information her doctor doesnt seem to be real interested in helping with that and the treatment shes on isnt working at all.

Hello, my father has had ulcerative colitis for about 20 years now. Yes, it does go into remission. For some people it doesn’t act up for years on end, for other people it is a daily battle. It depends on how bad you have it. Your friend is going to have to go in for what they call a colonoscopy. This is where they probe your colon with a scope. This will give them a good idea of how severe the disease is. There are treatments. There are steroids that can be taken and there are a lot of other meds out there that are good too without the side-effects of the steroids. Your friend will have to see a specialist so his/her doctor will have to help recommend someone to them. This disease can be annoying and it may require a bit of a lifestyle change but, for most people it is just a nuisance that have to deal with now and then. I hope this helps.